CHANGES IN THE SMALL INTESTINE IN PULMONARY FIBROSIS. THE BODY'S RESPONSE TO EXPERIMENTAL PULMONARY FIBROSIS

dc.contributor.authorHasanova D.A
dc.contributor.authorBarnoev A.I
dc.date.accessioned2025-12-31T15:36:44Z
dc.date.issued2022-12-26
dc.description.abstractIn this review, we discuss three broad areas which have been explored that may be responsible for the combination of altered lung fibroblasts, loss of alveolar epithelial cells, and excessive accumulation of ECM: inflammation and immune mechanisms, oxidative stress and oxidative signaling, and procoagulant mechanisms.
dc.formatapplication/pdf
dc.identifier.urihttps://scholarexpress.net/index.php/wbph/article/view/1863
dc.identifier.urihttps://asianeducationindex.com/handle/123456789/49179
dc.language.isoeng
dc.publisherScholar Express Journals
dc.relationhttps://scholarexpress.net/index.php/wbph/article/view/1863/1628
dc.rightshttps://creativecommons.org/licenses/by-nc-nd/4.0
dc.sourceWorld Bulletin of Public Health; Vol. 17 (2022): WBPH; 128-129
dc.source2749-3644
dc.subjectPulmonary fibrosis
dc.subjectimmune system
dc.subjectidiopathic pulmonary fibrosis
dc.titleCHANGES IN THE SMALL INTESTINE IN PULMONARY FIBROSIS. THE BODY'S RESPONSE TO EXPERIMENTAL PULMONARY FIBROSIS
dc.typeinfo:eu-repo/semantics/article
dc.typeinfo:eu-repo/semantics/publishedVersion
dc.typePeer-reviewed Article

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