SOD2 Gene Polymorphism (Ala16Val, rs4880) as a Predictor of Liver Cirrhosis in Chronic Hepatitis C: The Role of Oxidative Stress in Fibrosis Progression

dc.contributor.authorQaxarov Abduqaxar Nabijanovich
dc.contributor.authorAleynik Vladimir Alekseyevich
dc.contributor.authorVasilevskiy Eduard Aleksandrovich
dc.contributor.authorDadabayev Omonjon Tolibjonovich
dc.date.accessioned2026-03-07T20:51:40Z
dc.date.issued2026-03-05
dc.description.abstractOxidative stress is a universal mechanism of hepatocyte damage in chronic hepatitis C (CHC). Manganese superoxide dismutase (SOD2) is the key mitochondrial antioxidant enzyme. The Ala16Val polymorphism (rs4880) affects enzyme import into the mitochondrial matrix. This study aimed to evaluate the association of SOD2 Ala16Val with liver cirrhosis (LC) formation in CHC patients and to assess its relationship with disease severity markers and esophageal varices (EV)
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dc.identifier.urihttps://geniusjournals.org/index.php/emrp/article/view/7354
dc.identifier.urihttps://asianeducationindex.com/handle/123456789/118539
dc.language.isoeng
dc.publisherGenius Journals
dc.relationhttps://geniusjournals.org/index.php/emrp/article/view/7354/6053
dc.rightshttps://creativecommons.org/licenses/by-nc/4.0
dc.sourceEurasian Medical Research Periodical; Vol. 54 (2026): EMRP; 1-8
dc.source2795-7624
dc.subjectSOD2
dc.subjectAla16Val
dc.subjectrs4880
dc.titleSOD2 Gene Polymorphism (Ala16Val, rs4880) as a Predictor of Liver Cirrhosis in Chronic Hepatitis C: The Role of Oxidative Stress in Fibrosis Progression
dc.typeinfo:eu-repo/semantics/article
dc.typeinfo:eu-repo/semantics/publishedVersion
dc.typePeer-reviewed Article

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