Childhood cyanotic congenital heart disease and iron deficiency

dc.contributor.author	Nabeel Amer Mohammed Ali Al Quraishi
dc.contributor.author	Ali Jalil Malallah Al Rubaye
dc.contributor.author	Zainab Haleem Abed
dc.contributor.author	Asaad Hasan Noaman
dc.date.accessioned	2026-01-02T11:33:09Z
dc.date.issued	2023-06-10
dc.description.abstract	Inadequate tissue oxygenation results from cyanotic congenital heart disease. In an effort to increase red blood cell bulk, which will boost oxygen supply to the tissues, this in turn drives greater erythropoietin release from the kidneys. As a result of the prolonged erythropoiesis, polycythemia and frequently iron shortage arise from the depletion of iron reserves. Blood viscosity is enhanced and there is a propensity for bleeding thrombosis in both polycythemia and iron shortage
dc.format	application/pdf
dc.identifier.uri	https://geniusjournals.org/index.php/emrp/article/view/4451
dc.identifier.uri	https://asianeducationindex.com/handle/123456789/77639
dc.language.iso	eng
dc.publisher	Genius Journals
dc.relation	https://geniusjournals.org/index.php/emrp/article/view/4451/3780
dc.source	Eurasian Medical Research Periodical; Vol. 21 (2023): EMRP; 76-78
dc.source	2795-7624
dc.subject	bleeding
dc.subject	Inadequate
dc.subject	results
dc.subject	kidneys
dc.title	Childhood cyanotic congenital heart disease and iron deficiency
dc.type	info:eu-repo/semantics/article
dc.type	info:eu-repo/semantics/publishedVersion
dc.type	Peer-reviewed Article

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