DISEASES RESULTING FROM DISORDERS OF MELOTONIN HORMONES AND THEIR TREATMENT

dc.contributor.authorAbduvaliyeva Feruza Mukhammatnurovna
dc.contributor.authorFoziljonova Latofatxon Bakhtiyorjon qizi
dc.date.accessioned2025-12-29T17:59:00Z
dc.date.issued2025-05-29
dc.description.abstractThe hormone melatonin plays a vital role in regulating the body's biological clock mechanisms, controlling the sleep-wake cycle, and coordinating numerous physiological processes. Disruption in the secretion of this hormone, which is synthesized by the pineal gland, leads to the development of various pathological conditions. This article analyzes the effects of both melatonin deficiency and excess on the human body, the spectrum of diseases arising from these conditions, and modern treatment approaches. The research results indicate that melatonin disruptions not only impair the sleep-wake cycle but also contribute to the development of metabolic syndrome, weakened immune function, increased risk of oncological diseases, and psychoneurological disorders. Modern therapeutic approaches include individualized dosing regimens, treatments based on chronobiological principles, and comprehensive therapy strategies.
dc.formatapplication/pdf
dc.identifier.urihttps://webofjournals.com/index.php/5/article/view/4393
dc.identifier.urihttps://asianeducationindex.com/handle/123456789/24711
dc.language.isoeng
dc.publisherWeb of Journals Publishing
dc.relationhttps://webofjournals.com/index.php/5/article/view/4393/4753
dc.rightshttps://creativecommons.org/licenses/by-nc-nd/4.0
dc.sourceWeb of Medicine: Journal of Medicine, Practice and Nursing ; Vol. 3 No. 5 (2025): WOM; 580-584
dc.source2938-3765
dc.subjectmelatonin, pineal gland, circadian rhythm, sleep disorders, chronobiology, melatonin deficiency, metabolic syndrome, oncology, immunity, neuroendocrine system
dc.titleDISEASES RESULTING FROM DISORDERS OF MELOTONIN HORMONES AND THEIR TREATMENT
dc.typeinfo:eu-repo/semantics/article
dc.typeinfo:eu-repo/semantics/publishedVersion
dc.typePeer-reviewed Article

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