Mitochondrial Dysfunction in Neurodegenerative Diseases

dc.contributor.authorAbdullayeva N.N
dc.contributor.authorKasimov A.A.
dc.contributor.authorSuyarova K. A.
dc.contributor.authorXanturayeva M. Z.
dc.date.accessioned2025-12-31T12:15:24Z
dc.date.issued2023-06-30
dc.description.abstractThis review discusses the following mechanisms damage and dysregulatory effects on mitochondria: activation of nonspecific response mechanisms to disruption of the native structure of proteins – unfolded protein response (UPR); loss of mitochondrial function proteins; direct toxic effects of defective proteins on mitochondria; mitochondrial activation PGK mechanisms; dysregulation of the processes of utilization, division and fusion of mitochondria; traffic disruption and intracellular distribution of mitochondria.
dc.formatapplication/pdf
dc.identifier.urihttps://scholarsdigest.org/index.php/ijsnms/article/view/313
dc.identifier.urihttps://asianeducationindex.com/handle/123456789/44870
dc.language.isoeng
dc.publisherScholars Digest Publishing
dc.relationhttps://scholarsdigest.org/index.php/ijsnms/article/view/313/305
dc.sourceInternational Journal of Studies in Natural and Medical Sciences; Vol. 2 No. 6 (2023); 94-103
dc.source2949-8848
dc.source2949-8953
dc.subjectmitochondria, neurodegenerative processes, programmed cell death, endoplasmic reticulum stress. Key words: mitochondria, neurodegenerative processes, programmed cell death, stress of endoplasmic reticulum.
dc.titleMitochondrial Dysfunction in Neurodegenerative Diseases
dc.typeinfo:eu-repo/semantics/article
dc.typeinfo:eu-repo/semantics/publishedVersion
dc.typePeer-reviewed Article

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